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FB2026_01
,
released March 12, 2026
FlyBase curator comment: this entry is used to capture phenotypic information when the particular allele (or allele combination) used by the author could not be determined but the context of the experiment suggests that the phenotype being described is some kind of loss of function.
When salmunspecified and salrunspecified are absent from the entire wing blade, the vein L2 does not form and the intervein regions L2/3 and L4/5 disappear, causing an anterior shift in the L3 and L5 veins.
Neuronal clones derived from single central nervous system precursor cells taken from salm null embryos and cultured in vitro have thinner and poorly branched fibres compared to wild-type clones. The mutant clones are often surrounded by cell debris and their branches form "anastomosing" structures, which is not seen in wild-type clones. The clones are comprised of neuronal and glial cells. Tubulin does not reach the growth cone in mutant neurons.
In salm mutant embryos, there is no dorsal trunk and the dorsal branch often bifurcates to form an anterior and a posterior branch. This appears to be a transformation from dorsal branch to lateral trunk identity. A transformation of dorsal trunk to visceral branch cells is also seen.
No obvious defects are seen in mutant eye discs.
Embryos show partial transformation of the labium to prothorax and of segment A9 to A8.
salmunspecified has lethal | embryonic stage phenotype, non-suppressible by salaXX
salmunspecified has abdominal lateral pentascolopidial chordotonal organ lch5 | increased number phenotype, enhanceable by aosΔ7
salmunspecified is an enhancer of abdominal lateral pentascolopidial chordotonal organ lch5 | increased number phenotype of aosΔ7
salmunspecified argosΔ7 double mutant embryos have a dramatic chordotonal organ overproduction phenotype that is more severe than that seen in either single mutant. 9 or more lateral chordotonal organs per hemisegment are frequently seen, that are often (as in salmunspecified single mutants) scattered in lateral and dorsal positions. The double mutant embryos lack oenocytes.