Homozygous clones in the fifth adult abdominal segment show transformation to a more anterior identity, as shown by the loss of the black pigmentation characteristic of this segment. The effect is cell autonomous. The average size and frequency of homozygous clones induced at the cellular blastoderm stage are not significantly reduced in any body segments relative to control clones. Homozygous clones in the anterior compartment of the first leg show a reduction in the transverse row of bristles and the number of sex comb teeth. Clones in the first leg can also show an apical bristle (a characteristic of the second leg) at the distal end of the tibia. Clones in the second leg have no phenotypic abnormalities. Clones in the third leg show abnormalities, particularly in the distal leg structures; the femur and tibia are slightly abnormal, while the tarsal segments are severely distorted. All five tarsal segments are present but are truncated and have a hooked shape. Homozygous kisS embryos derived from homozygous kisS female germline clones (lacking both maternal and zygotic kis function) have a similar, but less severe phenotype than homozygous kis1 embryos derived from homozygous kis1 female germline clones.
Spradling.
Revertable at high frequency in the presence of P-element transposase.
kisS is revertible to wild type upon providing P\T function, indicating that the P{ry11} insertion causes the kisS mutation.