FB2026_01 , released March 12, 2026
FB2026_01 , released March 12, 2026
Aberration: Dmel\Df(1)sc19
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General Information
Symbol
Df(1)sc19
Species
D. melanogaster
Name
FlyBase ID
FBab0009479
Feature type
chromosomal_deletion
Also Known As
Df(1)sc19, sc19
Computed Breakpoints include
Features within 1A8--1B4
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Progenitor
Tp(1;2)sc19
Mutagen
segregation
Class of aberration (relative to wild type)
chromosomal_deletion
Class of aberration (relative to progenitor)
Breakpoints
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data
Genetic mapping information
Comments
Comments on Cytology
Sequence Crossreferences
DDBJ / EMBL / GenBank
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location (0)
    If no genes are listed here, it may be because the affected region is very large. The JBrowse insert above may show an error for the same reason, and other FlyBase tools such as CytoSearch may also fail for large regions. You can contact FlyBase for more help.
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    Phenotypic Data
    In combination with other aberrations
    NOT in combination with other aberrations

    Homozygous and hemizygous embryos have a week muscle phenotype, which is only slightly different from that of wild-type embryos.

    (Duan et al., 2007)

    The number of neurons in the dorsal organ (DO) ganglion are reduced in mutant embryos compared to wild type. The number of olfactory receptor neurons in the DO ganglion is normal in the mutant embryos.

    (Grillenzoni et al., 2007)

    Df(1)sc19 clones produce normal olfactory sensilla on the surface of the third antennal segment.

    (Reddy et al., 1997)

    Embryonic muscles 27 and 18 are only rarely affected. But in 26% of the mutant hemisegments muscle 5 is abnormally formed or displaced. Muscle 25 is often abnormal or missing.

    (Carmena et al., 1995)

    Hemizygous clones in the labellum lack all taste bristles; the taste pegs as well as the pseudotracheal structures are absent.

    (Ray and Rodrigues, 1994)

    Hemizygous embryos were examined with polarised light microscopy and antibody staining: muscle pattern incomplete due to muscle absences.

    (Drysdale et al., 1993)

    Df(1)sc19 embryos show a partial hypoplasy of the CNS.

    (Martin-Bermudo et al., 1993)

    Homozygous females show a strong loss of tergite chaetae.

    (Mari-Beffa et al., 1991)

    Gaps in the ventral cord and occasional clusters of cells are missing from the PNS, revealed by antibody staining for neuroblasts.

    (Cabrera et al., 1987)

    homozygous lethal Homozygous germline clones produce viable cells.

    (Garcia-Bellido and Robbins, 1983)
    Stocks (3)
    Notes on Origin
    Discoverer
     
    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     

    50-89% of Df(1)sc19/+ adults lack posterior supraalar bristles

    (Wrischnik et al., 2003)

    The lethal phenotype can be rescued partially by the P{3.2T3} and more fully by the P{5.9T3} constructs.

    (Martin-Bermudo et al., 1993)
    Synonyms and Secondary IDs (5)
    References (30)